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performed re- designed research; J.-s.D. The ability to induce IL‐6 from DC was preserved in cell lysates from MHC‐II −/− mice immunized with OVA . However, IL‐6 induction was significantly reduced when the lysates were extracted from mice deficient in CD8 T cells as a result of direct genetic knockout (CD8 −/−) or β2m −/− . Such responses can occur through direct binding to MHC class II (MHC II)–expressing tumor cells, or indirectly via activation of professional antigen-presenting cells (APC) that take up and present the tumor antigen. Then, they secrete cytokines to induce other cells in the immune system including B cells, CD8 T cells, and macrophages to trigger an immune response. In addition, the expression of MHC class II-restricted antigens by tumour cells is required at the site of successful rejection, indicating that activation of CD4 + T cells must also occur in the tumour microenvironment. Responsive T Cells : Present antigen to cytotoxic T cell lymphocytes (CD8+ T Cells); Present antigen to helper T cell lymphocytes; (CD4+ T cells). Co-receptor responsible : Binds with CD8 coreceptors molecules on cytotoxic T cells Tumor-specific CD4+ T cells have been shown to mediate efficient antitumor immune responses against cancer. This means that MHC contains a CD8-binding site. Major histocompatibility complex (MHC) class II–associated invariant chain (Ii) plays a critical role in antigen presentation, forming MHC class II peptide complexes for the generation of CD4 + T cell responses. Therefore, it occasionally causes false positive signals between MHC tetramers and CD8 … CD4 T cells are helper T cells with CD4 glycoprotein on their cell surface as the T cell receptor. They recognize antigens presented along with the MHC class II molecules by antigen-presenting cells. From gated CD 11b hi cells --> 70 % MHC II hi CD11 C + ... As I understand it, naive Cd8 T cells have high expression of CD62, effector/memory Cd8 T cells express CD44 highly. MHC Class I present 8-10 amino acid peptides: MHC Class II presents 14-18 amino acid peptides. Author contributions: J.-s.D., R.L.F., and B.M. MHC II −/ CD8 T cells were cotransferred into the same recipients, MHC II −/ CD8 T cells still expanded better than WT cells (Fig. 1B), indicating that the enhanced proliferation of MHC II −/ CD8 T cells is a cell-intrinsic property.
CD8 is known to bind MHC to assist in TCR and MHC binding.